Stress deficits in reward behaviour are associated with and replicated by dysregulated amygdala-nucleus accumbens pathway function in mice
Abstract Reduced reward interest/learning and reward-to-effort valuation are distinct, common symptoms in neuropsychiatric disorders for which chronic stress is a major aetiological factor.Glutamate neurons in basal amygdala (BA) project to various regions including nucleus accumbens (NAc).The BA-NAc neural pathway is activated by reward and aversi